Sex-specific epi-marks stop girl foetuses from being masculinised when they experience atypically high testosterone, and vice versa for boy foetuses.
Different epi-marks protect different sex-specific traits from being masculinised or feminised – some affect the genitals, others sexual identity, and yet others affect sexual partner preference.
However, when these epi-marks are transmitted across generations from fathers to daughters or mothers to sons, they may cause reversed effects, such as the feminisation of some traits in sons, such as sexual preference, and similarly a partial masculinisation of daughters.
The study solves the evolutionary riddle of homosexuality, finding that ‘sexually antagonistic’ epi-marks, which normally protect parents from natural variation in sex hormone levels during foetal development, sometimes carry over across generations and cause homosexuality in opposite-sex offspring.
The mathematical modeling demonstrates that genes coding for these epi-marks can easily spread in the population because they always increase the fitness of the parent but only rarely escape erasure and reduce fitness in offspring.
‘Transmission of sexually antagonistic epi-marks between generations is the most plausible evolutionary mechanism of the phenomenon of human homosexuality,’ said study co-author Sergey Gavrilets, a professor at the University of Tennessee-Knoxville.
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]]>It is used internationally by men who suffer from;
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Call: 097 954 6725
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